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|本期目录/Table of Contents|

缺血损伤和第二信使系统(PDF)

《心脏杂志》[ISSN:1009-7236/CN:61-1268/R]

期数:
1996年第3期
页码:
162-166
栏目:
综述
出版日期:
1996-05-01

文章信息/Info

Title:
-
作者:
楚英杰X 　综述　孙济川　田清海　审校
西安医科大学　西安　710004
Author(s):
-
关键词:
心肌缺血;  第二信使学说
Keywords:
-
分类号:
-
DOI:
-
文献标识码:
-
摘要:
-
Abstract:
-

参考文献/References

［1］Campbell AK. The investigation of intracellular Ca as a regulator. Campbell AK, ed. Intracellu-lar calcium : Its universal role as regulator. New York: John Wiley & Sons Ltd, 1993: 23.

［2］ 周衍椒, 张镜如, 主编. 生理学. 北京: 人民卫生出版社, 1991: 53.

［3］Teo TS, Wang JH. Mechanism of activation of a cyclic adenosine 3′,5′monophosphate phosphodiesterase from bovine heart by calcium ions. J Biol Chem 1973; 248 (17) : 5950.

［4］ 连秀峰, 赵东科, 牛小麟, 等, 主编. 钙拮抗剂的药理与临床. 西安: 陕西人民教育出版社, 1991:39～ 40.

［5］周　林. 三磷酸肌醇和甘油三酯在冠心病高血压发病中的作用. 心血管病学进展1993; 14 (5) :293.

［6］ 王迪浔, 主编. 病理生理学. 北京: 人民卫生出版社, 1994: 389～ 390.

［7］西田俊郎, 他. 缺血损害の分子机制. 代谢1987;25 (5) : 3.

［8］ Flack M , Sowers JR. Epidemiologic and clinical aspects of insulin resistance and hyperinsu-linemia. Am J Med 1991; 91 (1A ) : 11S.

［9］Das DK, Engelman RM , Prasad MR, et al. Im-provement of ischemia-reperfusion-induced my-ocardial dysfunction by modulating calcium-overload using a novel, specific calmodulin antagonist CGS 9343B. Biorchem Pharmacol 1989;1: 38 (8) : 465.

［10］Beresewicz A. Antagonists of calmodulin delay injury development in the severely ischemic perfused working rat heart.Acta Physiol Pol 1988; 39 (4) : 225.

［11］Boddeke HW , Willfert B, Hugtenburg JG, et al. Anti-ischemic activity of various calmodulin antagonists. Pharmacology 1988; 37 (4) : 240.

[12]Meerson FZ, Malyshev HU , Larionov NP, et al. Prevention of ischemic and reperfusion ar-rhythmias using the calmodulin blocker trifluoperazine. Kardiolog ia 1988; 28 (5) : 91.

[13]　Frass O , Sharma HS, Knoll R, et al. Enhanced gene expression of calcium regulatory protiens in stunned porcine myocardium. Cardiovasc Res 1993; 27 (11) : 2073.

[14]Yoshida K, Ogura Y, Wakasugi C. Histochem-ical study of calmodulin in dog myocardial is-chemia and sudden cardiac death. J pn Heart J 1992; 33 (5) : 717.

[15]Mouton R, Genade S, Boschmans SA , et al.The role of alpha 1-adrenergic stimulation in-insitol phosphate metabolism during post-ischemic reperfusion. L if e S ci 1992; 51 (26) : 2033.

[16]Mouton R, Genade S, Huisamen B, et al. The effect of ischaemic-reperfusion on [3H] inositol phosphates and ins (1, 4, 5) P3 levels in cardiac atria and ventricles—a comparative study. Mol Cell Biochem 1992; 115 (2) : 195.

[17] Thandroyen FT , Muntz KH, Bula LM , et al.A lterations in beta-adrenergic receptors adeny-late cyclase and cyclic AMP concentrations during acute myocardial ischemia and reperfusion. Circulation 1990; 82 (3 supp 1) : 1130.

[18]Szilvassy Z, Jakab I, Bor P, et al. Cicletanine attenuates overdrive pacing induced global my-ocardial ischemia in rabbits: possible role of cardiac cyclic nucleotides. Coron Artery Dis1993; 4 (5) : 443.

[19]Saman S, Coetze WA , Opie LH. Inhibition by simulated ischemia or hypoxia of after-depolar-izations provoked by cyclic AM P: significance for ischemic and reperfusion arrhythmias. J Mol Cell Cardiol 1988; 20 (2) : 91.

[20]Lubbe WF, Podzuweit T , Opie LH. Potential arrhythmogenic role of cyclic adenosine monophosphate(AMP) and calcium overload:implications for prophylactic effects of beta-blackers in myocardial infarction and proar-rhythmic effects of phosphodiesterase in-hibitors. J Am Coll Cardiol 1992; 19 (7) : 1622.

[21]Eckel J , Asskamp B, Reinaner H. Induction of insulin resistance in primary cultured adult car-diacmyocytes. Endocrinology 1991; 129 (1) : 345.

备注/Memo

备注/Memo:
收稿 1995-03-25；修回 1995-09-08
更新日期/Last Update: 1996-05-01