可能因为您的浏览器不支持样式,您可以更新您的浏览器到最新版本,以获取对此功能的支持,访问下面的网站,获取关于浏览器的信息:
[1]Siess W. Molecular mechanism of platelet activation. Physiol Rev , 1989; 69: 58.
[2]朱智明, 瞿文生. 血小板的活化与活化的血小板. 医学与哲学,1994; 1: 24.
[3]Lefer AM. Salntary action of thromboxane synthesis inhibition during global myocardial ischemia. Naunyn Schmiedebergs Arch Pharmacol, 1982; 321: 130.
[4]Schror K, Thiemermann C. Treatment of acute myocardial ischemia with a selective antagonist of thromboxane receptors. Br J P harmacol, 1986; 87: 631.
[5]Smyth DW ,Martin JF. Influence of platelet size before coronary angioplasty on subsequent restenosis. Eur J Clin Invest, 1993;23: 361.
[6] Radomski MW , Palmer RMJ , Moncada S. An Larginine/nitric oxide pathway present in human platelets regulates aggregation.Proc Natl Acad Sci USA , 1990; 87: 5193.
[7] Ej Langford. Inhibition of platelet activity by S-nitrosoglutathione during coronary angioplasty. Lancet, 1994; 344: 1458.
[8]池田久雄, 冷牟田浩司.血栓の生化学诊断. Mebio, 1994; 5: 51.
[9]阮长耿.血栓与止血.江苏省金陵科技著作出版社, 1994; 65.
[10]Gulba DC, Barthels M , Reil GH, et al. Thrombin/antithrombin Ⅲ complex level as early predicter of reocculusion after successful thrombalysis. Lancet, 1988; Ⅱ (8602) : 97.
[11]Rapold HJ , Kuemmerli H,Weiss M , et al. Monitoring of fibrin generation during thrombolytic therapy of acute myocardial infarction with recomhinant tissue-type plasminogen activator.Circu lation, 1989; 79: 980.
[12]Sakamoto T, Yasue H,Ogawa H, et al. Association of patency of the infarct-related coronary artery with plasma levels of plasminogen activator inhibitor activity in acute myocardial infarction. Am J Cardiol, 1992; 70: 271.