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|本期目录/Table of Contents|

心肌细胞凋亡与心室重构的关系(PDF)

《心脏杂志》[ISSN:1009-7236/CN:61-1268/R]

期数:
2006年第1期
页码:
95-96,99
栏目:
综述
出版日期:
2006-01-01

文章信息/Info

Title:
-
作者:
杨永曜 综述 李隆贵 审校
第三军医大学新桥医院心内科, 重庆 400037
Author(s):
-
关键词:
心肌细胞凋亡心室重构
Keywords:
-
分类号:
R331.31;R542.22
DOI:
-
文献标识码:
A
摘要:
近几年心肌凋亡和心室重构的关系及防治有一些重要进展 ,作者对此方面相关新进展作一综述。
Abstract:
-

参考文献/References

[1] 代政学, 张玉顺. 心肌细胞凋亡与心血管疾病[J]. 心脏杂志,2002, 14 (4):347-348.

[2]Jang YM, Kendaiah S, Drew B,et al. Doxorubicin treatment in vivo activates caspase12 mediated cardiac apoptosis in both male and female rats?[J]. FEBS Letters, 2004,577:483-490.

[3]Kong JY, Rabkin W. Cytoskeletal actin degradation induced by lovastatin in cardiomyocytes is mediated through caspase2?[J]. Cell Biol Int, 2004, 28:781-790.

[4]Philipp S, Pagel I, Hhnel K, et al. Regulation of caspase3 and Fas in pressure overloadinduced left ventricular dysfunction[J]. Eur J Heart Fail,2004,6:845-851.

[5]Sallee D, Qiu Y, Liu J, et al.Fas ligand gene transfer to the embryonic heart induces programmed cell death and outflow tract defects[J]. Dev Biol,2004,267:309-319.

[6]Dzimiri N, AlBahnasi K, AlHalees Z. Myocardial hypertrophy is not a prerequisite for changes in early gene expression in left ventricular volume overload[J]. Fundam Clin Pharmacol, 2004 ,18(1):39-44.

[7]Qin F, Liang MC, Liang CS. Progressive left ventricular remodeling, myocyte apoptosis, and protein signaling cascades after myocardial infarction in rabbits[J]. Biochim Biophys Acta, 2005,1740(3):499-513.

[8]Lancel S, Petillot P, Favory R, et al. Expression of apoptosis regulatory factors during myocardial dysfunction in endo toxemic rats[J]. Crit Care Med,2005,33(3):492-496.

[9]Peker C, SardaMantel L, Loiseau P. Imaging apoptosis with 99mTcannexinV in experimental subacute myocarditis[J]. J Nucl Med,2004 ,45(6):1081-1086.

[10]Mak KM,Wen K,RenC,et al. Dilinoleoylphosphatidylcholine reproduces the antiapoptotic actions of polyenylphosphatidylcholine against ethanolinduced hepatocyte apoptosis[J]. Alcohol Clin Exp Res 2003,27(6):997-1005.

[11]Kitaoka H,Kuwahara T,Nishida K,et al.Hypertrophic cardiomyopathy with progression from apical hypertrophy to asymmetrical septal hypertrophy: a case report[J]. J Cardiol,2005,45(4):155-159.

[12]Wencker D, Chandra M, Nguyen K, et al. A mechanistic role for cardiac myocyte apoptosis in heart failure[J]. J Clin Invest, 2003,111:1497 -1504.

[13]Balsam LB, Kofidis T, Robbins RC.Caspase3 inhibition preserves myocardial geometry and longterm function after infarction[J]. J Surg Res, 2005 ,124(2):194-200.

[14]Hayakawa Y, Chandra M, Miao W, et al. Inhibition of cardiac myocyte apoptosis improves cardiac function and abolishes mortality in the peripartum cardiomyopathy of Galpha(q) transgenic mice[J]. Circulation, 2003,108:3036-3041.

[15]Zhao ZQ, Morris CD, Budde JM, et al.Inhibition of myocardial apoptosis reduces infarct size and improves regional contractile dysfunction during reperfusion[J]. Cardiovasc Res,2003,59:132-142.

[16]Baines CP, Molkentin JD. Stress signaling pathways that modulate cardiac myocyte apoptosis[J]. J Mol Cell Cardiol, 2005,38:47-62.

备注/Memo

备注/Memo:
收稿日期:2005-07-25.基金项目:贵州省科技攻关项目资助(黔科通2004-40)通讯作者:李隆贵,教授,博士生导师,主要从事心肌病和心力衰竭方面的研究 Email:lgli602@hotmail.com 作者简介:杨永曜,博士生 Tel:13640537519 Email:yvuy11@tom.com
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