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[1] Kusachi S, Ninomiya Y. Myocardial infarction and cardiac fibro genesis. Fibrogenesis, cellular and molecular basis[M]. Razzaque Eurekah Press,2003.
[2] Cossu G, Bianco P. Mesoangioblastsvascular progenitors for extravascular mesodermal tissues[J]. Curr Opin Genet Dev ,2003,13(5):537 -542.
[3] Camelliti P, Green CR, LeGrice I,et al. Fibroblast network in rabbit sinoatrial node: structural and functional identification of homogeneous and heterogeneous cell coupling[J]. Circ Res ,2004,94(6):828- 835.
[4] Goldsmith EC, Hoffman A, Morales MO, et al. Organization of fibroblasts in the heart[J]. Dev Dyn,2004,230(4):787 -794.
[5] Gaudesius G, Miragoli M, Thomas SP, et al. Coupling of cardiac electrical activity over extended distances by fibroblasts of cardiac origin[J]. Circ Res ,2003,93(5):421 -428.
[6] Kohl P. Heterogeneous cell coupling in the heart: an electrophysio logical role for fibroblasts[J]. Circ Res,2003,93(5):381- 383
[7] Camelliti P, Devlin GP, Matthews KG, et al. Spatially and temporally distinct expression of fibroblast connexins after sheep ventricular infarction[J]. Cardiovasc Res ,2004,62(2):415- 425.
[8] Tao ZY, Cavasin MA, Yang F, et al.Temporal changes in matrix metalloproteinase expression and inflammatory response associated with cardiac rupture after myocardial infarction in mice[J].Life Sci ,2004,74(12):1561 -1572.
[9] Lindsey ML. MMP induction and inhibition in myocardial infarction[J]. Heart Fail Rev,2004,9(1):7-19.
[10]Turner NA, Porter KE, Smith WH, et al. Chronic beta2adrenergic receptor stimulation increases proliferation of human cardiac fibroblasts via an autocrine mechanism[J]. Cardiovasc Res,2003,57(3):784-792.
[11]顾俊,王长谦 .基质金属蛋白酶与冠脉再狭窄 [J] .心脏杂志,2005,17(1):77-88.
[12]Chiquet M, Renedo AS, Huber F, et al. How do fibroblasts translate mechanical signals into changes in extracellular matrix production? [J].Matrix Biol ,2003,22(1):73–80.
[13]Papakrivopoulou J, Lindahl GE, Bishop JE, et al. Differential roles of extracellular signalregulated kinase 1/2 and p38MAPK in mechanical loadinduced procollagen alpha1(I) gene expression in cardiac fibroblasts[J]. Cardiovasc Res ,2004,61(4):736 -744.
[14]Kamkin A, Kiseleva I, Isenberg G, et al. Cardiac fibroblasts and the mechanoelectric feedback mechanism in healthy and diseased hearts[J]. Prog Biophys Mol Biol, 2003,82(1-3):111- 120.