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[1]谢元博,陈 娟,李 巍.雾霾重污染期间北京居民对高浓度PM2.5持续暴露的健康风险及其损害价值评估[J].环境科学,2014,35(1):1-8.
[2]郭新彪,魏红英.大气PM2.5对健康影响的研究进展[J].科学通报,2013,58(13):1171-1177.
[3]中国环境保护部.GB3095-2012《环境空气质量标准》[S].北京:中国环境科学出版社,2012.
[4]World Health Organization.Ambient(outdoor)air quality and health[M].Geneva:World Health Organization,2016.
[5]Boldo E,Medina S,Le Tertre A,et al.Apheis: Health impact assessment of long-term exposure to PM2.5 in 23 European cities[J].Eur J Epidemiol,2006,21(6):449-458.
[6]Franklin M,Zeka A,Schwartz J.Association between PM2.5 and all-cause and specific-cause mortality in 27 US communities[J].J Expo Sci Environ Epidemiol,2007,17(3):279-287.
[7]Huang W,Cao J,Tao Y,et al.Seasonal variation of chemical species associated with short-term mortality effects of PM(2.5)in Xi’an,a central city in China[J].Am J Epidemiol,2012,175(6): 556-566.
[8]Sun X,Wei H,Young DE,et al.Differential pulmonary effects of wintertime California and China particulate matter in healthy young mice[J].Toxicol Lett,2017,278:1-8.
[9]Zhang SY,Shao D,Liu H,et al.Metabolomics analysis reveals that benzoapyrene,a component of PM2.5,promotes pulmonary injury by modifying lipid metabolism in a phospholipase A2-dependent manner in vivo and in vitro[J].Redox Biol,2017,13:459-469.
[10]Qian Y,Castranova V,Shi X.New perspectives in arsenic-induced cell signal transduction[J].J Inorg Biochem,2003,96(2-3): 271-278.
[11]Olsen CE,Liguori AE,Zong Y,et al.Arsenic upregulates MMP-9 and inhibits wound repair in human airwayepithelial cells[J].Am J Physiol Lung Cell Mol Physiol,2008,295(2):L293-L302.
[12]Luevano J,Damodaran C.A review of molecular events of cadmium-inducedcarcinogenesis[J].J Environ Pathol Toxicol Oncol,2014,33(3):183-194.
[13]Li FJ,Surolia R,Li H,et al.Lowdose cadmium exposure induces peribronchiolar fibrosis through site-specific phosphorylation of vimentin[J].Am J Physiol Lung Cell Mol Physiol,2017,313(1):80-91.
[14]Cox JN,Rahman MA,Bao S,et al.Cadmium attenuates the macrophage response to LPS through inhibition ofthe NF-kB pathway[J].Am J Physiol Lung Cell Mol Physiol,2016,311(4):L754-L765.
[15]Ashraf U,Kanu AS,Mo Z,et al.Lead toxicity in rice:effects,mechanisms,and mitigation strategies--a mini review[J].Environmental science and pollution research international,2015,22(23):18318-18332.
[16]Kaczynska K,Walski M,Szereda-Przestaszewska M.Long-term ultrastructural indices of lead intoxication in pulmonary tissue of the rat[J].Microsc Microanal,2013,19(6):1410-1415.
[17]Boskabaddy MH,Farkhondeh T.Inhaled lead exposure affects tracheal responsiveness and lung inflammation in guinea pigs during sensitization[J].Biol Trace Elem Res,2013,154(3):363-371.
[18]Li Q,Hu X,Bai Y,et al.The oxidative damage and inflammatory response induced by lead sulfide nanoparticles in rat lung[J].Food Chem Toxicol,2013,60:213-217.
[19]Liu SK,Cai S,Chen Y,et al.The effect of pollutional haze on pulmonary function[J].J Thorac Dis,2016,8(1):41-56.
[20]Niranjan R,Thakur AK.The toxicological mechanisms of environmental soot (black carbon) and carbon black:focus on oxidative stress and inflammatory pathways[J].Front Immunol,2017,8:763.
[21]Hystad P,Deners PA,Johnson KC,et al.Long-term residential exposure to air pollution and lung cancer risk[J].Epidemiology,2013,24(5):762-772.
[22]徐映如,王丹侠,张建文,等.PM10和PM2.5危害、治理及标准体系的概况[J].职业与健康,2013,29(1):117-119.
[23]胡亚东,马 吕,张 扬,等.北京和保定地区大气细颗粒物中可培养细菌的种群特征[J].环境科学,2017,38(4):1327-1339.
[24]陈 雯,黄淑洁,戴思思,等.杭州市区夏秋季空气PM2.5微生物分布特点的初步研究[J].中国卫生检验杂志,2013,23(10): 2361-2364.
[25]牛佳玉,肖纯凌,陈冬梅,等.PM2. 5对高血压(SHR/NCrl)大鼠呼吸道菌群的影响[J].卫生研究,2016,45(4):648-652.
[26]李少平,牛云彤.人类咽后壁菌群组成的定盆研究及抗生素敏感性测定[J].中国公共卫生,1995,11(7): 297-298.