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|本期目录/Table of Contents|

心肌重构过程中间隙连接的变化(PDF)

《心脏杂志》[ISSN:1009-7236/CN:61-1268/R]

期数:
2002年第5期
页码:
435-437
栏目:
综述与讲座
出版日期:
2002-09-01

文章信息/Info

Title:
-
作者:
毛建斌 综述; 黄德嘉 审校
华西医科大学附属第一医院心内科,四川成都610041
Author(s):
-
关键词:
心肌重构; 间隙连接
Keywords:
-
分类号:
R542.23
DOI:
-
文献标识码:
A
摘要:
本文概述了心肌细胞间间隙连接的结构及其在心肌的分布,着重阐述了心肌重构过程中间隙连接的变化及形成原因。
Abstract:
-

参考文献/References

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[3] Matsushita T,Oyamada M,Fujimoto K,et al. Remodeling of cell-cell and cell-extracellular matrix interactions at the border zone of rat myocardial infarcts[J].Circ Res,1999,85(11):1046-1055.

[4] Peter NS,Coromilas J,Sever NJ. Disturbed connexin 43 gap junction distribution correlate with the location of reentrant circuits in the epicardial border zone of healing canine infarcts that cause ventricular tachycardia[J].Circulation,1997,95(4):988-996.

[5] Kaprielian KR,Gunning M,Dupont E,et al. Downregulation of immunodetectable connexin 43 and decreased gap junction size in pathogenesis of chronic hibernation in the human left ventricle[J]. Circulation, 1998, 97(7):651-660.

[6] Zhuang J,Yamada KA,Saffitz JE,et al. Pulsatile stretch remodels cell-to-cell communication in cultured myocytes[J]. Circ Res,2000,87(4):316-322.

[7] Uzzama M,Honjo H,Takagishi Y,et al. Remodeling of gap junctional coupling in hypertrophied right ventricles of rats with monocrotaline-induced pulmonary hypertension[J]. Circ Res, 2000,86(8):871-878.

[8] Saffitz JE,Schuesser RB,Yamada KA. Mechanisms of remodeling of gap junction distributions and development of anatomic substrate of arrhythmias[J].Cardiovasc Res,1999,42(2):309-317.

备注/Memo

备注/Memo:
收稿日期：2001-07-10.
更新日期/Last Update: 2002-09-01