可能因为您的浏览器不支持样式,您可以更新您的浏览器到最新版本,以获取对此功能的支持,访问下面的网站,获取关于浏览器的信息:
[1] Schillaci G, Verdecchia P, Porcellati C, et al. Continuous relation between left ventricular mass and cardiovascular risk in essential hypertension[J]. Hypertension, 2000, 35(2):580-586.
[2] Conrad CH, Brooks WW, Hayes JA, et al. Myocardial fibrosis and stiffness with hypertrophy and heart failure in the spontaneously hypertensive rat[J]. Circulation, 1995, 91(1):161-170.
[3] 孙宁玲,王鸿懿,陈源源,等. EPK表达及活化在自发性高血压大鼠心肌肥厚中作用的研究[J]. 高血压杂志, 2002, 10(4):352-355.
[4] Liu JJ, Peng L, Bradley CJ, et al. Increased apoptosis in the heart of genetic hypertension, associated with increased fibroblasts[J]. Cardiovasc Res, 2000, 45(3):729-735.
[5] Makino N, Sugano M, Otsuka S, et al. Molecular mechanism of angiotensin Ⅱ typeⅠand type Ⅱreceptors in cardiac hypertrophy of spontaneously hypertensive rats[J]. Hypertension, 1997, 30(4):796-802.
[6] 余国龙,梁晓秋,谢秀梅,等. 高血压大鼠心脏肥厚过程中心肌细胞凋亡心肌纤维化动态观察[J]. 高血压杂志, 2001, 9(4):322-325.
[7] Rosendorff C.The rein-angiotensin system and vascular hypertrophy[J]. J Am Coll Cardiol, 1996, 28(4):803-812.
[8] Brunner-La Rocca HP, Vaddadi G, Esler MD. Recent insight into therapy of congestive heart failure: focus on ACE inhibition and angiotensin-Ⅱantagonism[J]. JAmCollCardiol,1999,33(5):1163-1173.
[9] 边瓯,陈富荣,陈铁锋,等. 自发性高血压大鼠左室重构过程中肝细胞生长因子与心肌纤维化的相关研究[J]. 心脏杂志, 2005, 17(4):323-325.