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|本期目录/Table of Contents|

应激性心肌病发病机制的研究现状

《心脏杂志》[ISSN:1009-7236/CN:61-1268/R]

期数:
2014年第2期
页码:
229-232
栏目:
综述
出版日期:
2014-01-20

文章信息/Info

Title:
Current research on pathogenesis of stress cardiomyopathy
作者:
奚银艳 综述; 郭瑞威 审校
（成都军区昆明总医院心内科、昆明医科大学附属昆明总医院临床学院，云南 昆明 650032）
Author(s):
XI Yin-yan;  GUO Rui-wei
(Department of Cardiology, Kunming General Hospital, Chengdu Military Area Command & Clinical College, Affiliated Kunming General Hospital, Kunming Medical University, Kunming 650032, Yunnan, China)
关键词:
应激性心肌病; 发病机制; 研究现状
Keywords:
stress cardiomyopathy;  pathogenesis;  current research situation
分类号:
R542
DOI:
-
文献标识码:
A
摘要:
1990年，日本学者Dote及Satoh等报道心理或躯体应激状态可以诱发一种急性扩张性心肌病-应激性心肌病( stress cardiomyopathy，SCM)，亦称“Tako-tsudo”心肌病，随后结合其发病早期独有的心尖部收缩功能障碍，将其命名为左心室心尖部气球样变综合征。SCM的临床表现为一过性类似急性心肌梗死样的胸痛。其发病机制目前尚不清楚。本文主要从一般情况和发病机制两方面阐述近年来国内外SCM的研究现状。
Abstract:
In the early 1990s, Japanese scholars Dote and Satoh reported a unique, reversible cardiomyopathy that appeared to be precipitated by acute emotional stress. This syndrome was initially named stress cardiomyopathy (SCM) and was also called Tako-tsubo cardiomyopathy (TC). SCM has subsequently been referred to as apical ballooning syndrome and “broken heart” syndrome. The clinical manifestation of SCM is transient, acute-myocardial-infarction-like chest pain but its pathogenesis is not clear. This review mainly introduces current research on the pathogenesis of stress cardiomyopathy at home and abroad in recent years.

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备注/Memo

备注/Memo:
收稿日期：2013-07-11.
通讯作者：郭瑞威，副主任医师，主要从事冠心病介入研究 Email:grw771210@163.com
作者简介：奚银艳，硕士生Email：2295462032@qq.com
更新日期/Last Update: 2014-01-16