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|本期目录/Table of Contents|

剪切应力与动脉粥样硬化(PDF)

《心脏杂志》[ISSN:1009-7236/CN:61-1268/R]

期数:
2003年第1期
页码:
78-80,83
栏目:
综述
出版日期:
2003-01-01

文章信息/Info

Title:
-
作者:
张铭曾智
四川大学华西医院心内科,四川 成都 610041
Author(s):
-
关键词:
剪切应力动脉粥样硬化血管重塑
Keywords:
-
分类号:
R543.5
DOI:
-
文献标识码:
A
摘要:
血液在血管中连续不断的流动,对血管内皮细胞产生持续的剪切力。流动剪切力在动脉粥样硬化的病理和病理生理过程中起着重要的活跃作用,本文综述了近年来这方面的研究进展。
Abstract:
-

参考文献/References

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[6] Lum RM, Wiley LM, Barakat AI, et al. Influence of different forms of fluid shear stress on vascular endothelial TGF-beta1 mRNA expression [J] . Int J Mol Med, 2000, 5(6):635-641

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[15] 邢海燕, 李宏妹. 流体切应力下血管内皮单核细胞趋化蛋白-1的表达 [J] , 中华病理学杂志, 2001, 30(5):361-364.

[16] Bao X, Lu C, Frangos JA, et al. Temporal gradient in shear butnot steady shear stress induces PDGF-A and MCP-1 expression in endothelial cells: role of NO, NF kappa B, and egr-1 [J] . Arterioscler Thromb Vasc Biol, 1999, 19(4):996-1003.

[17] 徐红新, 李建军, 李庚山. 核因子-kB与动脉粥样硬化 [J] . 中国动脉粥样硬化杂志, 2001, 9(2):180-181.

[18] 陈友琴, 陈槐卿. 流体切应力对内皮细胞粘附分子表达的影响 [J] . 生物医学工程杂志, 2001, 18(2):201-520.

[19] Urbich C, Fritzenwanger M, Zeiher AM, et al. Laminarshear stress upregulates the complement-inhibitory protein clusterin: a novel potent defense mechanism against complement-induced endothelial cell activation [J] . Circulation, 2000, 101(4):352-355.

备注/Memo

备注/Memo:
收稿日期:2002-03-27.
更新日期/Last Update: 2003-01-01